By A. Masuda, T. Kobayashi, Y. Ohyabu (auth.), Richard L. Hughson, David A. Cunningham, James Duffin (eds.)
Impact of previous O2 respiring on Hypoxic Hypercapnic Ventilatory Responses in people; A. Masuda, et al. Inhibitory Dopaminergic Mechanisms Are useful in Peripherally Chemodenervated Goats; K.D.O'Halloran, et al. impression of eight Hours of Isocapnic/Poikilocapnic Hyopxia at the Ventilatory reaction to CO2; M. Fatemian, P.A.Robbins. Ventilatory Responses to Hypoxia after 6 Hours Passive Hyperventilation in people; X. Ren, P.A. Robbins. Ventilatory results of eight Hours of Isocapnic Hypoxia with and with no beta-Blockade; C.Clar, et al. Modulation of Ventilatory Sensitivity to Hypoxia through Dopamine and Domperiodone earlier than and After lengthy publicity to Hypoxia in Humans;M.E.F. Pedersen, et al. adjustments in breathing keep an eye on in the course of and After forty eight Hours of either Isocapnic and Poikilocapnic Hypoxia in people; J.G. Tansley, et al. Chemoreflex results of Low Dose Sevoflurane in people; J.J. Pandit, et al. Dynamics of the Cerebral Blood stream reaction to Sustained Euoxic Hypocapnia in people; M.J. Poulin, et al. proof for a valuable function of Protein Kinase C in Modulation of the Hypoxic Ventilatory reaction within the Rat; D. Gozal,et al. Synaptic Connections to Phrenic Motneurons within the Decerbrate Rat; G.-F. Tian, et al. 26 extra Articles. Index.
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Extra info for Advances in Modeling and Control of Ventilation
A: Before iontophoresis. B: After 45 minutes of chloride iontophoresis at \0 nA. In each example, trace I is the membrane potential and trace 2 is the phrenic nerve discharge. As apart of our attempts to determine the synaptic inputs responsible for the 3-stage pattern of membrane potential trajectories we used chloride iontophoresis to reverse inhibitory hyperpolarization to depolarisation in 58 phrenic motoneurons in order to observe the pattern of synaptic inhibition. In alt ofthem, the hyperpolarizing waves during late expiration were easily reversed to depolarising waves after about I minute.
J. Appl. Physiol. 33: 47-54. 1972. 9. D. G. Howson. A prediction-correction scheme for forcing alveolar gases along certain time courses. J. Appl. Physiol. 52: 1353-1357, 1982. 10. A. Hypoxie ventilatory decline: site of action. J. Appl. Physiol. 373-374. 11. , A. Dahan, L. LJ. van den Elsen, E. Olofsen, A. Berkenbosch, and J. van Kleef. Acute pain and central nervous system arousal do not restore impaired hypoxie ventilatory response during sevoflurane sedation. Anes/hesiology 85: 295-303, 1996.
Reddan, J. F. Grover, and J. Rankin. Effect of chronic exposure to hypoxia on ventilatory response to CO 2 and hypoxia. J. Appl. Physiol. 31: 586-592, 1971. 4. S. Milledge. Respiratory regulation in man during acclimatisation to high altitude. J. Physiol. 168: 631-643,1963. 5. C. M. c. Mithoefer. Adaptation to high altitude: respiratory response to CO2 and 02' J. Appl. Physiol. 6: 15S-162, 1953. 4 VENTILATORY RESPONSES TO HYPOXIA AFTER 6 HOURS PASSIVE HYPERVENTILATION IN HUMANS Xiaohui Ren and Peter A.
Advances in Modeling and Control of Ventilation by A. Masuda, T. Kobayashi, Y. Ohyabu (auth.), Richard L. Hughson, David A. Cunningham, James Duffin (eds.)